| General Information of MIC (ID: MIC01183) |
| MIC Name |
Shigella sonnei (enterobacteria)
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| MIC Synonyms |
Bacterium sonnei
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| Body Site |
Gut
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| Lineage |
Kingdom: Bacteria
Phylum: Proteobacteria
Class: Gammaproteobacteria
Order: Enterobacterales
Family: Enterobacteriaceae
Genus: Shigella
Species: Shigella sonnei
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| Oxygen Sensitivity |
Facultative anaerobe
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| Microbial Metabolism |
Fermentative; respiratory
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| Gram |
Negative
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| Host Relationship |
Opportunistic pathogen
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| Genome Size (bp) |
4949533
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| No. of Coding Genes |
4808
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| No. of Non-Coding Genes |
93
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| No. of Small Non-Coding Genes |
93
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| No. of Gene Transcripts |
4901
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| No. of Base Pairs |
4949533
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| Description |
Shigella sonnei is a non-motile, nonspore-forming, facultative anaerobic, Gram-negative bacterium. Its non-motile characteristic means that this species doesn have flagella to facilitate its movement like many other human enterobacteria.
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| External Links |
Taxonomy ID |
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| Genome Assembly ID |
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| GOLD Organism ID |
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| Host Immune Factors (HIFs) |
| Interleukin-1 beta |
| HIF ID |
HIFM0138 |
HIF Info
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Class |
Cytokine (Cyt) |
| Description |
Shigella sonnei infected cells was able to reduce pro-inflammatory immune responses such as IL-1Beta. |
[1] |
| C-X-C motif chemokine 8 |
| HIF ID |
HIFM0153 |
HIF Info
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Class |
Cytokine (Cyt) |
| Description |
Shigella sonnei could reduce pro-inflammatory immune responses such as IL-8. |
[1] |
| Tumor necrosis factor |
| HIF ID |
HIFM0226 |
HIF Info
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Class |
Cytokine (Cyt) |
| Description |
Shigella sonnei could reduce TNF-Alpha. |
[1] |
| Immunoglobulin M |
| HIF ID |
HIFM0266 |
HIF Info
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Class |
Immunoglobulin (Ig) |
| Description |
The abundance of Shigella sonnei is associated with IgM response. |
[2] |
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| Environmental Factor(s) |
| Disbiome ID |
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| gutMDisorder ID |
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| References |
| 1 |
The anti-apoptotic and anti-inflammatory effect of Lactobacillus acidophilus on Shigella sonnei and Vibrio cholerae interaction with intestinal epithelial cells: A comparison between invasive and non-invasive bacteria. PLoS One. 2018 Jun 6;13(6):e0196941. doi: 10.1371/journal.pone.0196941. eCollection 2018.
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| 2 |
IRAK-4 and MyD88 deficiencies impair IgM responses against T-independent bacterial antigens.Blood. 2014 Dec 4;124(24):3561-71. doi: 10.1182/blood-2014-07-587824. Epub 2014 Oct 15.
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